



Figures and tables from this article:
Table 1. Summary table of study characteristics. N = 30 studies (1995-2010).




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Figures and tables from this article:
Table 1. Summary table of study characteristics. N = 30 studies (1995-2010).
Figures and tables from this article:
Table 1. Summary table of study characteristics. N = 30 studies (1995-2010).
Figures and tables from this article:
Table 1. Summary table of study characteristics. N = 30 studies (1995-2010).
Figures and tables from this article:
Fig. 1. During NREM sleep, abnormal thalamocortical structures may be unable to generate sufficient slow oscillations to drive the reactivation of hippocampal memory traces. These same structures may also be unable to facilitate normal spindle activity, preventing efficient declarative memory consolidation due to an absence in cortical plastic changes. Decreases in spindle activity lead to failure in inhibiting sensory information from reaching the neocortex. Thus, the individual is awakened and kept awake by sensory information, consequently experiencing disturbed NREM sleep.View Within ArticleCopyright © 2011 Elsevier Ltd. All rights reserved.prs.rt('data_end');
Figures and tables from this article:
Fig. 1. During NREM sleep, abnormal thalamocortical structures may be unable to generate sufficient slow oscillations to drive the reactivation of hippocampal memory traces. These same structures may also be unable to facilitate normal spindle activity, preventing efficient declarative memory consolidation due to an absence in cortical plastic changes. Decreases in spindle activity lead to failure in inhibiting sensory information from reaching the neocortex. Thus, the individual is awakened and kept awake by sensory information, consequently experiencing disturbed NREM sleep.View Within ArticleCopyright © 2011 Elsevier Ltd. All rights reserved.prs.rt('data_end');
Figures and tables from this article:
Fig. 1. During NREM sleep, abnormal thalamocortical structures may be unable to generate sufficient slow oscillations to drive the reactivation of hippocampal memory traces. These same structures may also be unable to facilitate normal spindle activity, preventing efficient declarative memory consolidation due to an absence in cortical plastic changes. Decreases in spindle activity lead to failure in inhibiting sensory information from reaching the neocortex. Thus, the individual is awakened and kept awake by sensory information, consequently experiencing disturbed NREM sleep.View Within ArticleCopyright © 2011 Elsevier Ltd. All rights reserved.prs.rt('data_end');
Figures and tables from this article:
Table 1. Studies of sleep disturbances in children with ADHD with subjective methods.
Figures and tables from this article:
Table 1. Studies of sleep disturbances in children with ADHD with subjective methods.
Figures and tables from this article:
Table 1. Studies of sleep disturbances in children with ADHD with subjective methods.